[1] |
Kurtz S, Mowat F, Ong K, et al. Prevalence of primary and revision total hip and knee arthroplasty in the United States from 1990 through 2002[J]. J Bone Joint Surg Am, 2005, 87( 7 ): 1487-1497.
|
[2] |
Ravi B, Croxford R, Reichmann WM, et al. The changing demographics of total joint arthroplasty recipients in the United States and Ontario from 2001 to 2007[J]. Best Pract Res Clin Rheumatol, 2012, 26( 5 ): 637-647.
|
[3] |
Singh JA, Vessely MB, Scott Harmsen W, et al. A population-based study of trends in the use of total hip and total knee arthroplasty,1969-2008[J]. Mayo Clin Proc, 2010, 85( 10 ): 898-904.
|
[4] |
Sloan M, Premkumar A, Sheth NP. Projected volume of primary total joint arthroplasty in the U.S., 2014 to 2030[J]. J Bone Joint Surg Am, 2018, 100( 17 ): 1455-1460.
|
[5] |
Khan M, Osman K, Green G, et al. The epidemiology of failure in total knee arthroplasty:avoidingyournextrevision[J]. Bone Joint J,2016, 98-B( 1 Suppl A ): 105-112.
|
[6] |
Fehring TK, Odum S, Griffin WL, et al. Early failures in total knee arthroplasty[J]. Clin Orthop Relat Res, 2001( 392 ): 315-318.
|
[7] |
林丽能, 王辉, 孙效棠, 等. 一期和二期髋关节翻修术后感染复发率的Meta分析[J/CD]. 中华关节外科杂志( 电子版 ), 2019,13( 1 ): 73-77.
|
[8] |
Huang Y, Guan D, Li Y, et al. A systematic review and metaanalysis comparing complications following total hip arthroplasty for systemic lupus erythematosus versus for non-systemic lupus erythematosus[J/OL]. J Orthop Surg Res, 2022, 17( 1 ): 235.DOI:10.1186/s13018-022-03075-8.
|
[9] |
Xu D, Ding C, Cheng T, et al. Inflammatory bowel disease is associated with an increased risk of adverse events in patients undergoing joint arthroplasty[J]. Bone Joint Res, 2023, 12( 6 ):362-371.
|
[10] |
Newman JM, Sultan AA, Khlopas A, et al. What are the shortterm outcomes in multiple sclerosis patients after total knee arthroplasty?[J]. J Knee Surg, 2019, 32( 2 ): 165-170.
|
[11] |
Jiang W, Xu H, Wang X, et al. More complications and higher transfusion rate in patients with rheumatoid arthritis than osteoarthritis undergoing total hip arthroplasty[J]. Int Orthop, 2023,47( 5 ): 1189-1196.
|
[12] |
Zhang AR, Cheng QH, Yang YZ, et al. Meta-analysis of outcomes after total knee arthroplasty in patients with rheumatoid arthritis and osteoarthritis[J]. Asian J Surg, 2024, 47( 1 ): 43-54.
|
[13] |
Sleiman AG, Vallette N, Milto AJ, et al. The effect of autoimmune skin disorders on post-operative outcomes following arthroplasty[J/OL]. Surgeon, 2023, 21( 5 ): e292-e300. DOI:10.1016/j.surge.2023.03.003.
|
[14] |
Gold PA, Garbarino LJ, Ramkumar PN, et al. Psoriasis and postsurgical infections in primary total knee arthroplasty: an analysis of 10, 727 patients[J]. J Arthroplasty, 2022, 37( 8 ): 1575-1578.
|
[15] |
Smith GD, Ebrahim S. ‘Mendelian randomization’: can genetic epidemiology contribute to understanding environmental determinants of disease?[J]. Int J Epidemiol, 2003, 32( 1 ): 1-22.
|
[16] |
Ference BA, Ray KK, Catapano AL, et al. Mendelian randomization study of ACLY and cardiovascular disease[J]. N Engl J Med, 2019,380( 11 ): 1033-1042.
|
[17] |
Myers TA, Chanock SJ, Machiela MJ. LDlinkR: an R package for rapidly calculating linkage disequilibrium statistics in diverse populations[J/OL]. Front Genet, 2020, 11: 157. DOI:10.3389/fgene.2020.00157.
|
[18] |
Burgess S, Thompson SG. Use of allele scores as instrumental variables for Mendelian randomization[J]. Int J Epidemiol, 2013,42( 4 ): 1134-1144.
|
[19] |
Staley JR, Blackshaw J, Kamat MA, et al. PhenoScanner: a database of human genotype-phenotype associations[J]. Bioinformatics,2016, 32( 20 ): 3207-3209.
|
[20] |
Verbanck M, Chen CY, Neale B, et al. Detection of widespread horizontal pleiotropy in causal relationships inferred from Mendelian randomization between complex traits and diseases[J]. Nat Genet,2018, 50( 5 ): 693-698.
|
[21] |
Burgess S, Thompson SG. Interpreting findings from Mendelian randomization using the MR-Egger method[J]. Eur J Epidemiol,2017, 32( 5 ): 377-389.
|
[22] |
Milne RL, Kuchenbaecker KB, Michailidou K, et al. Identification of ten variants associated with risk of estrogen-receptor-negative breast cancer[J]. Nat Genet, 2017, 49( 12 ): 1767-1778.
|
[23] |
Hartwig FP, Davey Smith G, Bowden J. Robust inference in summary data Mendelian randomization via the zero modal pleiotropy assumption[J]. Int J Epidemiol, 2017, 46( 6 ): 1985-1998.
|
[24] |
Bowden J, Del Greco M F, Minelli C, et al. A framework for the investigation of pleiotropy in two-sample summary data Mendelian randomization[J]. Stat Med, 2017, 36( 11 ): 1783-1802.
|
[25] |
Yuan S, Bruzelius M, Damrauer SM, et al. Cardiometabolic, lifestyle,and nutritional factors in relation to varicose veins: amendelian randomization study[J/OL]. J Am Heart Assoc, 2021, 10( 21 ):e022286. DOI:10.1161/JAHA.121.022286.
|
[26] |
Durst C, Rajaee L, Chang K, et al. Psoriasis is a risk factor for surgical site infection after primary TKA[J]. Clin Orthop Relat Res,2024, 482( 7 ): 1196-1200.
|
[27] |
Zhang LJ. Type1 interferons potential initiating factors linking skin wounds with psoriasis pathogenesis[J/OL]. Front Immunol, 2019,10: 1440. DOI:10.3389/fimmu.2019.01440.
|
[28] |
Aly R, Maibach HE, Mandel A. Bacterialflorainpsoriasis[J]. Br J Dermatol, 1976, 95( 6 ): 603-606.
|
[29] |
Ouyang W. Distinct roles of IL-22 in human psoriasis and inflammatory bowel disease[J]. Cytokine Growth Factor Rev, 2010,21( 6 ): 435-441.
|
[30] |
Tohyama M, Yang L, Hanakawa Y, et al. IFN-α enhances IL-22 receptor expression in keratinocytes: apossible role in the development of psoriasis[J]. J Invest Dermatol, 2012, 132( 7 ):1933-1935.
|
[31] |
Tzu J, Kerdel F. From conventional to cutting edge: the New Era of biologics in treatment of psoriasis[J]. Dermatol Ther, 2008, 21( 2 ):131-141.
|
[32] |
Pätzold L, Stark A, Ritzmann F, et al. IL-17C and IL-17RE promote wound closure in a staphylococcus aureus-based murine wound infection model[J/OL]. Microorganisms, 2021, 9( 9 ): 1821.DOI:10.3390/microorganisms9091821.
|
[33] |
Ramirez-Carrozzi V, Sambandam A, Luis E, et al. IL-17C regulates the innate immune function of epithelial cells in an autocrine manner[J]. Nat Immunol, 2011, 12( 12 ): 1159-1166.
|
[34] |
Lauffer F, Jargosch M, Baghin V, et al. IL-17C amplifies epithelial inflammation in human psoriasis and atopic eczema[J]. J Eur Acad Dermatol Venereol, 2020, 34( 4 ): 800-809.
|
[35] |
Vandeghinste N, Klattig J, Jagerschmidt C, et al. Neutralization of IL-17C reduces skin inflammation in mouse models of psoriasis and atopic dermatitis[J]. J Invest Dermatol, 2018, 138( 7 ): 1555-1563.
|
[36] |
Johnston A, Fritz Y, Dawes SM, et al. Keratinocyte overexpression of IL-17C promotes psoriasiform skin inflammation[J]. J Immunol,2013, 190( 5 ): 2252-2262.
|
[37] |
Boehncke WH, Schön MP. Psoriasis[J]. Lancet, 2015, 386( 9997 ):983-994.
|
[38] |
Hernandez-Nicols BF, Robledo-Pulido JJ, Alvarado-Navarro A.Etiopathogenesis of psoriasis: integration of proposed theories[J].Immunol Invest, 2024, 53( 3 ): 348-415.
|
[39] |
Armstrong AW, Read C. Pathophysiology, clinical presentation, and treatment of psoriasis:areview[J]. JAMA, 2020, 323( 19 ): 1945-1960.
|
[40] |
Taylor-Williams O, Nossent J, Inderjeeth CA. Incidence and complication rates for total hip arthroplasty in rheumatoid arthritis:asystematic review and meta-analysis across four decades[J].Rheumatol Ther, 2020, 7( 4 ): 685-702.
|
[41] |
Ravi B, Croxford R, Hollands S, et al. Increased risk of complications following total joint arthroplasty in patients with rheumatoid arthritis[J]. Arthritis Rheumatol, 2014, 66( 2 ): 254-263.
|
[42] |
Cordtz R, Odgaard A, Kristensen LE, et al. Risk of medical complications following total hip or knee arthroplasty in patients with rheumatoid arthritis: a register-based cohort study from Denmark[J]. Semin Arthritis Rheum, 2020, 50( 1 ): 30-35.
|